15:20 - 16:50
Room: ANA Crowne Plaza “Ohtori” Room B
Workshop Session
Chair/s:
Ann Chen, Masaaki Murakami
Selective blockade of NLRP3 inflammasome by TCM in lupus nephritis
Ann Chen1, Shuk-Man Ka2, Feng-Cheng Liu3, Kuo-Feng Hua4, Shozo Izui5
1Department of Pathology, Tri-Service General Hospital, National Defense Medical Center, Taipei, Taiwan, 2Graduate Institute of Aerospace and Undersea Medicine, Academy of Medicine, National Defense Medical Center, Taipei, Taiwan, 3Department of Rheumatology/Immunology and Allergy, Department of Medicine, Tri-Service General Hospital, National Defense Medical Center, Taipei, Taiwan, 4Department of Biotechnology and Animal Science, National Ilan University, Ilan, Taiwan, 5Department of Pathology and Immunology, Faculty of Medicine, University of Geneva, Geneva, Switzerland

Lupus nephritis (LN) is one of the most common and severe complications in systemic lupus erythematous patients. The current treatments for LN mainly rely on glucocorticoids and other immunosuppressants. These drugs are partially effective and yet have considerable side effects, especially for long-term administration. Thus, new therapeutic approaches are clinically warranted for disease control. It has been shown that renal NLRP3 inflammasome activation play a key role in the pathogenesis of LN. Because LN results from autoimmune conditions, windows of opportunity exist for improving therapy by targeting NLRP3 inflammasome and its related molecular pathways. Traditional Chinese medicine (TCM) has been successfully used worldwide in treating various diseases, including autoimmune and renal conditions, with tremendously low toxicity and side effects, although individual mechanisms of action responsible for each of them to render beneficial effects are mostly vague.

In this report, we demonstrated that TCM-derived pure compounds exerted reno-protective effects in LN through regulating NLRP3 inflammasome, using our recently established screening platform with cell models, covering both immune cells and intrinsic renal cells, and a spontaneous mouse model of LN. Most of these tested components have been proved as of extremely low of cytotoxicity at a cell level, although further in vivo toxicology study is required for drug development purposes in the near future.


Reference:
Tu-WS10-2
Session:
Workshop 10, “Cytokines in autoimmune diseases”
Presenter/s:
Ann Chen
Presentation type:
Invited Speaker
Room:
ANA Crowne Plaza “Ohtori” Room B
Chair/s:
Ann Chen, Masaaki Murakami
Date:
Tuesday, 31 October 2017
Time:
15:26 - 15:36
Session times:
15:20 - 16:50