09:00 - 10:40
Room: Bibo BallroomAB
Invited & Short Lecture
Chair/s:
Leandros A Skaltsounis, Yang Ye
Pharmacological effects of novel cyclopropylacetylshikonin derivatives on melanoma cells
Nadine Kretschmer 1, Christin Durchschein 1, Beate Rinner 2, Alexander Stallinger 2, Alexander Deutsch 3, Birgit Lohberger 4, Antje Huefner 5, Rudolf Bauer 1
1 Institute of Pharmaceutical Science, Department of Pharmacognosy, University of Graz, Graz
2 Core Facility Alternative Biomodels and Preclinical Imaging, Medical University of Graz, Graz
3 Division of Hematology, Medical University of Graz, Graz
4 Department of Orthopedics and Trauma, Medical University Graz, Graz
5 Institute of Pharmaceutical Sciences, Department of Pharmaceutical Chemistry, University of Graz, Graz

According to the WHO, cancer is still a leading cause of morbidity and the second leading cause of death worldwide. The incidence of melanoma is rising faster than any other solid tumor type and melanoma are responsible for 79% of all skin cancer deaths. Especially at an advanced stage, melanoma is still one of the most aggressive and incurable types of cancer. Today, about 79% of all approved anti-cancer drugs are natural products or derived from them demonstrating their important role in medicine [1]. In a previous study, shikonin derivatives isolated from the roots of Onosma paniculata Bur. & Franch. (Boraginaceae) have emerged as interesting candidates for finding new drug leads against cancer [2]. We now prepared several novel semisynthetic shikonin derivatives and analyzed their cytotoxic potential against different melanoma cell lines. The most active compound was (R)-1-(1,4-dihydro-5,8-dihydroxy-1,4-dioxonaphthalen-2-yl)-4-methylpent-3-enyl cyclopropylacetate (CP). It was especially more active against two melanoma cell lines derived from metastatic lesions compared to the most active isolated derivative b - b -dimethylacrylshikonin (DMAS). IC50 values of CP and DMAS were 4.9 µM (WM164) and 3.2 µM (MUG-Mel-2), and 8.3 µM and 7.2 µM, respectively. Further investigations revealed that CP induced apoptosis, but did not lead to cell cycle arrest. Moreover, the ApoToxGlo® assay together with the LDH assay revealed that CP did not significantly damage the cell membrane up to 48h and 5.0 µM.

Acknowledgement: The Austrian Science Fund (FWF) is acknowledged for financial support (Project P27505).

References:
[1] Newman D. J., Cragg G. M (2016) Natural Products as Sources of New Drugs from 1981 to 2014. J Nat Prod 79(3): 629-661.
[2] Kretschmer N. Rinner B., Deutsch A.J., Lohberger B., Knausz H., Kunert O., Blunder M., Boechzelt H., Schaider H., Bauer R.(2012) Naphthoquinones from Onosma paniculata Induce Cell-Cycle Arrest and Apoptosis in Melanoma Cells. J Nat Prod, 75(5): 865-869.


Reference:
Session 1-3-SL-04:
Session:
Session 1-3:Natural products chemistry and drug discovery
Presenter/s:
Nadine Kretschmer
Presentation type:
Short lecture (oral presentation)
Room:
Bibo BallroomAB
Chair/s:
Leandros A Skaltsounis, Yang Ye
Date:
Wednesday, 29th August, 2018
Time:
09:55 - 10:10
Session times:
09:00 - 10:40